The neurologic phenotype in SCA8 BAC expansion but not BAC control traces shown the pathogenicity on the (CTG-CAG)n enlargement. In addition, the expression of noncoding (CUG)n enlargement ATXN8OS transcripts and the discovery of intranuclear polyglutamine inclusions prompt that SCA8 pathogenesis consists of poisonous obtain-of-perform mechanisms a
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